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Why does menstruation abdomen ache? The reason that schoolgirl menstruation stomachache

Researchers have finally found clues to explain aunt pain and premenstrual syndrome that afflict women in general.

So far, in the latest such studies, scientists have found a link between inflammatory biomarkers and the severity of premenstrual syndrome, which means that acute inflammation can cause all spasms and bloating.

For years, researchers have known that anti-inflammatory drugs can greatly relieve dysmenorrhea, and most doctors have advised patients to take anti-inflammatory drugs to calm their angry uterus.

But the study is the first large-scale study to confirm the biological link between inflammation and premenstrual syndrome. Although it's only just beginning, one day this research will help scientists find more effective treatments than the over-the-counter painkillers we're using now.

But that's all scientists have found - a team at the University of California, Davis, who surveyed 2939 American women and found a positive correlation between the severity of PMS and the presence of a high-sensitivity C-reactive protein.

High sensitivity C-reactive protein is a biomarker of inflammation in the body, so the researchers believe that the more this protein is in a person, the more inflammatory it is likely to be, and the more severe premenstrual syndrome (except for headaches).

"Premenstrual emotional symptoms, abdominal spasm, back pain, appetite gain, weight gain, abdominal distension, and breast distension (except headache) were positively correlated with the increase of biomarker high-sensitivity C-reactive protein of inflammation. Even after adjusting for a variety of confounding variables, there was a moderate positive correlation between the two, "the researchers reported in the Journal of women's health.

This is important because about 80% of women are said to have PMS. However, there are few studies on its causes or possible therapies. The best treatment we have now is to take the over-the-counter painkiller ibuprofen, or a prescription anti-inflammatory drug when the situation is very serious.

A new device also claims to be able to 'turn off' menstrual pain electronically, but it certainly can't stop premenstrual syndrome at the same time.

This means that millions of women have little choice but to endure monthly discomfort including cramps, nausea, vomiting and depression for most of their lives.

If we can find more information about the biological factors that contribute to PMS, we may have an opportunity to develop better treatments, or at least identify which biological factors are more dangerous. Understanding the link between inflammation and premenstrual syndrome may also lead scientists to the next step.

"The results also show that the factors associated with premenstrual syndrome are complex, suggesting that there may be different mechanisms for the etiology of some symptoms & hellip; & hellip; inflammation plays a key role in most PMS, although further longitudinal studies are needed to examine the relationship," the team wrote.

However, advising women to avoid inflammation related behaviors may help prevent dysmenorrhea, and anti-inflammatory drugs may also be effective in treating dysmenorrhea symptoms.

It's a long way to go. We've only made a preliminary attempt, but we can't suppress our inner joy. Now please allow me to ignore my great aunt until scientists come up with a new research method. Will she visit again?